The interaction mechanism involves altered absorption pathways and changes in redox balance
Major Ingredients Water, Triethylhexanoin, Hydrogenated Poly(C6-14 Olefin), Butylene Glycol, Niacinamide, Methylpropanediol, 1,2-Hexanediol, Phenyl Trimethicone, Diethoxyethyl Succinate, Polyglyceryl-10 Myristate, Borago Officinalis Extract, Centaurea Cyanus Flower Extract, Chamomilla Recutita (Matricaria) Flower/Leaf Extract, Lavandula Angustifolia (Lavender) Flower Water, Hyacinthus Orientalis (Hyacinth) Extract, Salvia Sclarea (Clary) Extract, Ethylhexylglycerin, Hibiscus Esculentus Fruit Extract, Corchorus Olitorius Leaf Extract, Carum Petroselinum (Parsley) Extract, Hexyl Cinnamal, Limonene, Linalool, Fragrance, Hippophae Rhamnoides Oil, Allantoin, Hydrogenated Lecithin, Sodium Gluconate, Glutathione, Adenosine, Cellulose Gum, Melia Azadirachta Leaf Extract, Melia Azadirachta Flower Extract, Curcuma Longa (Turmeric) Root Extract, Ocimum Sanctum Leaf Extract, Corallina Officinalis Extract, Biotin, Biotinoyl Tripeptide-1, Acetyl Hexapeptide-8, Tripeptide-1, Tripeptide-3, Myristoyl Pentapeptide-17, Hexapeptide-9, Gluconolactone, Alpha-Arbutin, Thioctic Acid Ingredients subject to change at manufacturer's discretion
10.1038/s41556-018-0178-0 Summary Keywords ferroptosis, programmed cell death, dermatology, cancer, inflammation Citation Liu L, Lian N, Shi L, Hao Z and Chen K (2023) Ferroptosis: Mechanism and connections with cutaneous diseases
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The data reported that IGF-1 significantly increased total cross-sectional area (tt.Ar.), cortical bone area ( Ct.Ar.), and cortical thickness (Ct.Th) in female KIR mice
